Oxidative stress involvement in chemically induced differentiation of K562 cells

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Oxidative stress involvement in chemically induced differentiation of K562 cells.

The erythroid differentiation of K562 cells could be achieved by exposure to several pharmacologic agents, including hemin, butyric acid (BA), and anthracycline antitumor drugs such as aclarubicin (ACLA) and doxorubicin (DOX). When used at subtoxic concentrations, these drugs induce the overexpression of erythroid genes, leading to hemoglobinization of cells. Because anthracyclines are known to...

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Oxidative stress in K562 cells differentiation 1 OXIDATIVE STRESS INVOLVEMENT IN CHEMICALLY-INDUCED DIFFERENTIATION OF K562 CELLS

The erythroid differentiation of K562 cells could be achieved by exposure to several pharmacological agents including hemin, butyric acid (BA) and anthracycline antitumor drugs such as aclarubicin (ACLA) and doxorubicin (DOX). When used at subtoxic concentration, these drugs induce the overexpression of erythroid genes leading to hemoglobinization of cells. Since anthracyclines are known to gen...

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Inhibition by 1,25 dihydroxyvitamin D3 of chemically induced erythroid differentiation of K562 leukemia cells.

The physiologically active form of vitamin D, 1,25 dihydroxyvitamin D3 [1,25(OH)2D3], was found to inhibit erythroid differentiation of human leukemic K562 cells. Differentiation was induced by 1 mumol/L arabinocytosine (Ara-C), 40 mumol/L tiazofurin, 1 mumol/L aphidicolin, or 1 mumol/L hydroxyurea, and was monitored daily by the appearance of hemoglobin in an increasing proportion of cells. Pr...

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Involvement of p38 kinase in hydroxyurea-induced differentiation of K562 cells.

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Antidiabetic effect of honey feeding in noise induced hyperglycemic rat: involvement of oxidative stress

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ژورنال

عنوان ژورنال: Free Radical Biology and Medicine

سال: 2000

ISSN: 0891-5849

DOI: 10.1016/s0891-5849(99)00195-1